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SLE is a universal disease with an augmented risk of early death and, if left untreated, will cause irreversible damage to the affected tissue, causing complications in the affected individuals
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This proteasomal inhibiting role can lead to a reduced proteasomal elimination of ubiquinated P-Iκ (phosphorylated-inhibitor kappa), inhibiting nuclear translocation of NF-κ, suppressing NF-κB activation and its consequences of tumor necrosis factor alpha (TNF-α), and NO production (51, 52)
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